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PARKIN is not required to sustain OXPHOS function in adult mammalian tissues - npj Parkinson's Disease

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PARKIN is not required to sustain OXPHOS function in adult mammalian tissues - npj Parkinson's Disease

Loss-of-function variants in the PRKN gene encoding the ubiquitin E3 ligase PARKIN cause autosomal recessive early-onset Parkinson's disease (PD). Extensive in vitro and in vivo studies have reported that PARKIN is involved in multiple pathways of mitochondrial quality control, including mitochondrial degradation and biogenesis. However, these findings are surrounded by substantial controversy due to conflicting experimental data.

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PARKIN is not required to sustain OXPHOS function in adult mammalian tissues - npj Parkinson's Disease

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PARKIN is not required to sustain OXPHOS function in adult mammalian tissues - npj Parkinson's Disease

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