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RIOK3 sustains colorectal cancer cell survival under glucose deprivation via an HSP90α-dependent pathway - Oncogenesis

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RIOK3 sustains colorectal cancer cell survival under glucose deprivation via an HSP90α-dependent pathway - Oncogenesis

Glucose oxidation via the pentose phosphate pathway serves as the primary cellular mechanism for generating nicotinamide adenine dinucleotide phosphate (NADPH). The central regions of solid tumors typically experience glucose deficiency, emphasizing the need for sustained NADPH production crucial to tumor cell survival. This study highlights the crucial role of RIOK3 in maintaining NADPH production and colorectal cancer (CRC) cell survival during glucose deficiency.

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RIOK3 sustains colorectal cancer cell survival under glucose deprivation via an HSP90α-dependent pathway - Oncogenesis

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RIOK3 sustains colorectal cancer cell survival under glucose deprivation via an HSP90α-dependent pathway - Oncogenesis

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