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OASL phase condensation induces amyloid-like fibrillation of RIPK3 to promote virus-induced necroptosis - Nature Cell Biology

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OASL phase condensation induces amyloid-like fibrillation of RIPK3 to promote virus-induced necroptosis - Nature Cell Biology

RIPK3–ZBP1–MLKL-mediated necroptosis is a proinflammatory cell death process that is crucial for antiviral host defence. RIPK3 self-oligomerization and autophosphorylation are prerequisites for executing necroptosis, yet the underlying mechanism of virus-induced RIPK3 activation remains elusive. Interferon-inducible 2′-5′ oligoadenylate synthetase-like (OASL) protein is devoid of enzymatic function but displays potent antiviral activity.

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OASL phase condensation induces amyloid-like fibrillation of RIPK3 to promote virus-induced necroptosis - Nature Cell Biology

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OASL phase condensation induces amyloid-like fibrillation of RIPK3 to promote virus-induced necroptosis - Nature Cell Biology

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