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Sphingolipid subtypes differentially control proinsulin processing and systemic glucose homeostasis - Nature Cell Biology

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Sphingolipid subtypes differentially control proinsulin processing and systemic glucose homeostasis - Nature Cell Biology

Impaired proinsulin-to-insulin processing in pancreatic β-cells is a key defective step in both type 1 diabetes and type 2 diabetes (T2D) (refs. 1,2), but the mechanisms involved remain to be defined. Altered metabolism of sphingolipids (SLs) has been linked to development of obesity, type 1 diabetes and T2D (refs. 3–8); nonetheless, the role of specific SL species in β-cell function and demise is unclear.

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Sphingolipid subtypes differentially control proinsulin processing and systemic glucose homeostasis - Nature Cell Biology

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Sphingolipid subtypes differentially control proinsulin processing and systemic glucose homeostasis - Nature Cell Biology

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