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E-cadherin interacts with EGFR resulting in hyper-activation of ERK in multiple models of breast cancer - Oncogene

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E-cadherin interacts with EGFR resulting in hyper-activation of ERK in multiple models of breast cancer - Oncogene

The loss of intercellular adhesion molecule E-cadherin is a hallmark of the epithelial-mesenchymal transition (EMT), during which tumor cells transition into an invasive phenotype. Accordingly, E-cadherin has long been considered a tumor suppressor gene; however, E-cadherin expression is paradoxically correlated with breast cancer survival rates.

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E-cadherin interacts with EGFR resulting in hyper-activation of ERK in multiple models of breast cancer - Oncogene

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E-cadherin interacts with EGFR resulting in hyper-activation of ERK in multiple models of breast cancer - Oncogene

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