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TREM1 disrupts myeloid bioenergetics and cognitive function in aging and Alzheimer disease mouse models - Nature Neuroscience

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TREM1 disrupts myeloid bioenergetics and cognitive function in aging and Alzheimer disease mouse models - Nature Neuroscience

Human genetics implicate defective myeloid responses in the development of late-onset Alzheimer disease. A decline in peripheral and brain myeloid metabolism, triggering maladaptive immune responses, is a feature of aging. The role of TREM1, a pro-inflammatory factor, in neurodegenerative diseases is unclear. Here we show that Trem1 deficiency prevents age-dependent changes in myeloid metabolism, inflammation and hippocampal memory function in mice.

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TREM1 disrupts myeloid bioenergetics and cognitive function in aging and Alzheimer disease mouse models - Nature Neuroscience

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TREM1 disrupts myeloid bioenergetics and cognitive function in aging and Alzheimer disease mouse models - Nature Neuroscience

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