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Transcription–replication conflicts underlie sensitivity to PARP inhibitors - Nature

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Transcription–replication conflicts underlie sensitivity to PARP inhibitors - Nature

An important advance in cancer therapy has been the development of poly(ADP-ribose) polymerase (PARP) inhibitors for the treatment of homologous recombination (HR)-deficient cancers1–6. PARP inhibitors trap PARPs on DNA. The trapped PARPs are thought to block replisome progression, leading to formation of DNA double-strand breaks that require HR for repair7.

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Transcription–replication conflicts underlie sensitivity to PARP inhibitors - Nature

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Transcription–replication conflicts underlie sensitivity to PARP inhibitors - Nature

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