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Selenium abandons selenoproteins to inhibit ferroptosis rapidly - Nature Metabolism

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Selenium abandons selenoproteins to inhibit ferroptosis rapidly - Nature Metabolism

Selenium is usually incorporated into selenoproteins, with important functions in redox regulation. A new study in Nature Metabolism reveals a previously unappreciated role for selenium-based chemical species as direct electron donors to reduce ubiquinone, thus contributing to redox homeostasis by preventing lipid peroxidation.

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Selenium abandons selenoproteins to inhibit ferroptosis rapidly - Nature Metabolism

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Selenium abandons selenoproteins to inhibit ferroptosis rapidly - Nature Metabolism

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