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Tolerable glycometabolic stress boosts cancer cell resilience through altered N-glycosylation and Notch signaling activation - Cell Death & Disease

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Tolerable glycometabolic stress boosts cancer cell resilience through altered N-glycosylation and Notch signaling activation - Cell Death & Disease

Chronic metabolic stress paradoxically elicits pro-tumorigenic signals that facilitate cancer stem cell (CSC) development. Therefore, elucidating the metabolic sensing and signaling mechanisms governing cancer cell stemness can provide insights into ameliorating cancer relapse and therapeutic resistance.

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Tolerable glycometabolic stress boosts cancer cell resilience through altered N-glycosylation and Notch signaling activation - Cell Death & Disease

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Tolerable glycometabolic stress boosts cancer cell resilience through altered N-glycosylation and Notch signaling activation - Cell Death & Disease

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