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TREM2 macrophage promotes cardiac repair in myocardial infarction by reprogramming metabolism via SLC25A53 - Cell Death & Differentiation

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TREM2 macrophage promotes cardiac repair in myocardial infarction by reprogramming metabolism via SLC25A53 - Cell Death & Differentiation

Efferocytosis and metabolic reprogramming of macrophages play crucial roles in myocardial infarction (MI) repair. TREM2 has been proven to participate in phagocytosis and metabolism, but how it modulates myocardial infarction remains unclear. In this study, we showed that macrophage-specific TREM2 deficiency worsened cardiac function and impaired post-MI repair.

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TREM2 macrophage promotes cardiac repair in myocardial infarction by reprogramming metabolism via SLC25A53 - Cell Death & Differentiation

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TREM2 macrophage promotes cardiac repair in myocardial infarction by reprogramming metabolism via SLC25A53 - Cell Death & Differentiation

Rate This Post

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Unlocking the Power of Knowledge for Humanity's Progress

Unlocking the Power of Knowledge
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Unlocking the Power of Knowledge
for Humanity's Progress