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Apoptotic stress causes mtDNA release during senescence and drives the SASP - Nature

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Apoptotic stress causes mtDNA release during senescence and drives the SASP - Nature

Senescent cells drive age-related tissue dysfunction partially through the induction of a chronic senescence-associated secretory phenotype (SASP)1. Mitochondria are major regulators of the SASP; however, the underlying mechanisms have not been elucidated2. Mitochondria are often essential for apoptosis, a cell fate distinct from cellular senescence. During apoptosis, widespread mitochondrial outer membrane permeabilization (MOMP) commits a cell to die3. Here we

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Apoptotic stress causes mtDNA release during senescence and drives the SASP - Nature

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Apoptotic stress causes mtDNA release during senescence and drives the SASP - Nature

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