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MLL3 loss drives metastasis by promoting a hybrid epithelial–mesenchymal transition state - Nature Cell Biology

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MLL3 loss drives metastasis by promoting a hybrid epithelial–mesenchymal transition state - Nature Cell Biology

Phenotypic plasticity associated with the hybrid epithelial–mesenchymal transition (EMT) is crucial to metastatic seeding and outgrowth. However, the mechanisms governing the hybrid EMT state remain poorly defined. Here we showed that deletion of the epigenetic regulator MLL3, a tumour suppressor frequently altered in human cancer, promoted the acquisition of hybrid EMT in breast cancer cells.

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metastasis cells hybrid epithelial mesenchymal cell biology biology microbiology

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MLL3 loss drives metastasis by promoting a hybrid epithelial–mesenchymal transition state - Nature Cell Biology

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MLL3 loss drives metastasis by promoting a hybrid epithelial–mesenchymal transition state - Nature Cell Biology

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