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Non-canonical β-adrenergic activation of ERK at endosomes - Nature

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Non-canonical β-adrenergic activation of ERK at endosomes - Nature

G-protein-coupled receptors (GPCRs), the largest family of signalling receptors, as well as important drug targets, are known to activate extracellular-signal-regulated kinase (ERK)—a master regulator of cell proliferation and survival1. However, the precise mechanisms that underlie GPCR-mediated ERK activation are not clearly understood2–4. Here we investigated how spatially organized β2-adrenergic receptor (β2AR) signalling controls ERK.

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Non-canonical β-adrenergic activation of ERK at endosomes - Nature

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Non-canonical β-adrenergic activation of ERK at endosomes - Nature

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